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Cognitive EnhancementPreclinical

Dihexa

HGF/MET signaling amplifier and among the most potent synaptogenic nootropics studied — note: foundational study retracted April 2025

Research Reality Check

Not Enough Evidence YetInteresting idea, but proof is still thin.
ClaimSome people claim Dihexa has clear value for cognitive enhancement research.
RealityMost support is early or indirect, so human results are not settled.
Bottom LineUse the evidence score, sources, and safety notes before taking any claim seriously.
Why People Believe ThisSimple explanations and user stories can sound more certain than the research is.
Watch Out For
Guaranteed resultsExact protocols presented as provenAnecdotes used as proof
234Discussions
2Citations

Evidence Dossier

62Evidence

Preclinical

Evidence score reflects source depth, citations, and research maturity. It is not a medical recommendation.

2Citations
234Discussions
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Dihexa at a glance

A fast read for beginners, with evidence strength, route context, safety depth, and community activity surfaced before the deeper sections.

Evidence score62Preclinical
Primary routeCognitive EnhancementRoute availability varies by context
Safety depthExperimentalReview safety notes before making assumptions
Community questions234Related discussions and experiences

Dihexa (N-hexanoic-Tyr-Ile-(6) aminohexanoic amide) is a small peptide-derived compound developed at Washington State University by Joseph Harding and colleagues. It was designed as a stable, orally bioavailable analog of angiotensin IV that potently amplifies hepatocyte growth factor (HGF) signaling through its receptor MET.

How It Works

HGF/MET signaling is critical for synaptogenesis - the formation of new synaptic connections - and has been implicated in cognitive enhancement and recovery from neurological injury. In animal models, Dihexa showed potency millions of times greater than BDNF in promoting synaptic formation, producing dramatic improvements in spatial learning and memory tasks.

However, the cognitive research community should note that the primary foundational paper (Bhatt DL, et al.) was retracted in April 2025 following concerns about data integrity. This does not eliminate all evidence for Dihexa's mechanism, but it significantly reduces the evidentiary basis for its most dramatic claimed effects. Use with informed caution; the compound's safety profile in humans has not been established through clinical trials.

Key Benefits

Synaptogenesis and synaptic density
Memory formation and recall
Cognitive enhancement
Potential neuroprotection