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MetabolicPreclinical

5-Amino-1MQ

Small molecule NNMT inhibitor that activates NAD+ metabolism, stem cell function, and metabolic gene programs

Research Reality Check

Not Enough Evidence YetInteresting idea, but proof is still thin.
ClaimSome people claim 5-Amino-1MQ has clear value for metabolic research.
RealityMost support is early or indirect, so human results are not settled.
Bottom LineUse the evidence score, sources, and safety notes before taking any claim seriously.
Why People Believe ThisSimple explanations and user stories can sound more certain than the research is.
Watch Out For
Guaranteed resultsExact protocols presented as provenAnecdotes used as proof
143Discussions
2Citations

Evidence Dossier

78Evidence

Preclinical

Evidence score reflects source depth, citations, and research maturity. It is not a medical recommendation.

2Citations
143Discussions
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5-Amino-1MQ at a glance

A fast read for beginners, with evidence strength, route context, safety depth, and community activity surfaced before the deeper sections.

Evidence score78Preclinical
Primary routeMetabolic EnhancerRoute availability varies by context
Safety depthExperimentalReview safety notes before making assumptions
Community questions143Related discussions and experiences

5-Amino-1MQ is a small membrane-permeable molecule that inhibits nicotinamide N-methyltransferase (NNMT), an enzyme expressed in adipose tissue that consumes SAM (S-adenosylmethionine) and methyl donors to methylate nicotinamide - effectively draining both NAD+ precursors and the methyl pool. By inhibiting NNMT, 5-Amino-1MQ redirects nicotinamide back toward NAD+ synthesis and restores the methyl pool, activating SIRT1, AMPK, and other longevity-associated metabolic regulators.

How It Works

In preclinical studies, 5-Amino-1MQ reduced adipocyte differentiation, activated fat-burning gene programs, and reduced body fat in diet-induced obese mice without changes in food intake. It also activated quiescent muscle stem cells (satellite cells), suggesting potential for muscle maintenance and regeneration. Unlike NAD+ precursors (NMN, NR) which simply flood the precursor pool, NNMT inhibition takes a more targeted approach by removing the drain on the system.

5-Amino-1MQ is orally bioavailable, which represents a practical advantage over most longevity peptides. It remains a research compound with no human clinical trials. Its mechanism is well-characterized at the cellular level but systemic effects in humans are not established.

Key Benefits

Fat loss via NNMT inhibition
NAD+ pathway activation
Stem cell (satellite cell) activation
Metabolic gene program restoration
SIRT1 and AMPK activation